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Our animated video is a must watch before your first open colonic session. Our open colonic device introduces purified warm water into the colon and allows water and waste to exit in a safe and comfortable way.

Then push the call button. Ultra clean,safe, sterile and effective. The water has the potential to reach the cecum or beginning of the ascending colon which can allow for a deeper cleanse.

Colonics may remove fecal matter from lower bowel. For Best Results the Industry Standard recommends three sessions within a week, or three days back to back.

Our professional certified colon hydrotherapists are committed to set the standard in colon hydrotherapy today. Your therapist is there throughout your entire session.

This can be done on lunch break. Normally clients can continue their normal day after, although some may need to stay closer to a toilet after.

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There is no offensive smell or mess to worry about with the Closed system. Its a full service experience. The treatment usually lasts 45 minutes on the table.

Your modesty is protected throughout the process. Once the comfort level is reached by the guest the water is released and flows through a disposable waste line.

True or false? Bacteria living in the same space, like the mouth, have evolved collaborations so generous that they are not possible with outside bacteria.

That was long held to be true, but in a new, large-scale study of microbial interactions, the resounding answer was "false.

Bacteria from the oral microbiome , by contrast, generally shared food and assistance more stingily with gum infector Aggregatibacter actinomycetemcomitans , or Aa for short.

Like many bacteria known for infections they can cause—like Strep—Aa often live peacefully in the mouth, and certain circumstances turn them into infectors.

The researchers and their sponsors at the National Institutes of Health would like to know more about how Aa interacts with other microbes to gain insights that may eventually help fight acute periodontitis and other ailments.

The findings are surprising because bacteria in a microbiome have indeed evolved intricate interactions making it seem logical that those interactions would stand out as uniquely generous.

Some mouth microbes even have special docking sites to bind to their partners, and much previous research has tightly focused on their cooperations.

The new study went broad. We thought they would not interact well with the other bugs, but it was the opposite," Whiteley said. The study's scale was massive.

Researchers manipulated and tracked nearly all of Aa's roughly 2, genes using an emergent gene tagging technology while pairing Aa with 25 other microbes—about half from the mouth and half from other body areas or the environment.

They did not examine the mouth microbiome as a whole because multi-microbial synergies would have made interactions incalculable.

Instead, the researchers paired Aa with one other bug at a time—Aa plus mouth bacterium X, Aa plus colon bacterium Y, Aa plus dirt fungus Z, and so on.

They published their results in the Proceedings of the National Academy of Sciences. The researchers looked at each of Aa's genes necessary for survival while it infected a mouse—when Aa was the sole infector, when it partnered with a fellow mouth bacterium and when paired with a microbe from colon, dirt, or skin.

The other microbe was giving Aa things that it needed, so it didn't have to make them itself. To understand "transposon sequencing," picture a transposon as a DNA brick that cracks a gene, breaking its function.

The brick also sticks to the gene and can be detected by DNA sequencing, thus tagging that malfunction.

Every Aa bacterium in a pile of 10, had a brick in a random gene. If Aa's partner bacterium, say, E.

Aa surviving with more broken genes meant a partner microbe was giving it more assistance. Aa bacteria with broken genes that a partner could not compensate for were more likely to die, reducing their count.

It very likely does have them, but the study's results point to not all relationships being cooperative.

Some microbiomes could have high fences and share sparsely. Smoking, poor hygiene, or diabetes—all associated with gum disease—might be damaging defensive microbiomes and allowing outside bacteria to help Aa attack gum tissue.

It's too early to know that, but Whiteley's lab wants to dig deeper, and the research could have implications for other microbiomes. Explore further.

The brick also sticks to the gene and can be detected by DNA sequencing, thus tagging that malfunction.

Some microbiomes could have high fences and share sparsely. Any findings, conclusions or recommendations are those of the authors and not necessarily those of the National Institutes of Health.

Bacteria behind acute periodontitis. Aggregatibacter actinomycetemcomitans most often lives peacefully in the mouth until circumstances lead it to become infectious.

It forms flower-like colonies, here under a microscope sporting a colorful stain added by a researcher. Aa is gram-negative.

Aggregatibacter actinomycetemcomitans breaths oxygen, but in some partnerships with other microbes, it was tested in an anoxic environment.

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